AML Muscle Growth

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AML Muscle Growth


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AML Muscle Growth
Post-Workout Stack

Cutting-edge research reveals that consuming certain
compounds immediately after weight training can
increase muscular growth and strength. One newly
developed product from Advanced Molecular Labs,
AML Muscle GrowthPost-Workout Stack, is comprised
of ingredients meant to enhance muscle growth, lean
body mass, strength, power and recovery like no other
post-workout product ever has before. AML Muscle
Growth Post-Workout Stack
works because it consists
of the powerful muscle-building compounds beta-hydroxy-
beta-methylbutryrate (HMB) (3,000 milligrams), phosphatidic
acid (750 milligrams) and vitamin D3 (4,000 IUs). And this
exceptional mixture of compounds in this mighty post-workout
stack was designed to precisely target the function of specific
protein molecules within the body, triggering an anabolic
environment that will generate massive muscle growth.
Here's how it works:

Hmb And Muscle Protein Breakdown

HMB positively influences muscle growth by attenuating muscle protein breakdown instead of stimulating muscle protein synthesis. In a study showing the influence of HMB on muscle protein breakdown, it was found that 3,000 milligrams of HMB increased muscle protein synthesis rather weakly, but attenuated muscle protein breakdown by a whopping 57 percent. Importantly, a second study showed that the ability of HMB to reduce muscle protein catabolism does actually increase muscle growth and strength, when combined with resistance training.

Vitamin D For Muscle Growth

Vitamin D is a powerful prohormone that drives muscle growth by regulating the expression of many different genes within the human genome. Some of the muscle-promoting genes regulated by vitamin D are involved in the production and function of testosterone.

Another gene controlled by vitamin D enhances insulin sensitivity, in part, by increasing expression of the insulin receptor gene. The increase in insulin receptor expression results in more insulin receptors on the surface of the muscle cell, which causes a more sensitive signaling response to insulin. Because insulin function boosts muscle growth, the enhanced insulin response brought on by vitamin D can, in due course, deliver more muscle growth.

Some more recent scientific evidence has also shown the capacity of vitamin D3 to increase insulin receptor levels within the muscle cell, providing a more robust anabolic response to leucine intake. A study by Salles et al. looked at the influence that vitamin D3- the most active form of vitamin D in humans- had on leucine-activated insulin signaling in muscle cells. Since muscle protein synthesis is positively regulated by leucine consumption, the researchers wanted the see if vitamin D3 could enhance leucine activation of the insulin pathway. The results of this investigation showed that vitamin D3 enhanced the ability of leucine to activate insulin signaling while also increasing the rate of muscle protein synthesis. This investigation also demonstrated that this increase in insulin
signaling and muscle protein production was due primarily to the
increased expression of the insulin receptor gene- which increased
the number and activity level of insulin receptors embedded within
the muscle cell, fostering a greater anabolic response within the
muscle cell.

Phosphatidic Acid For Strength
And Inceased Muscle Mass

Phosphatidic Acid (PA) is a phospholipid found in the cell membrane
involved in many different cell signaling cascades, including one
signaling pathway that stimulates muscle growth.

While it has clearly been established that mTOR is necessary for the
hypertrophic response to resistance training, the activation of mTOR
has previously been proposed to occur through the IGF-1 signaling
cascade. However, several recent scientific studies conclusively
illustrate that an increase in PA is responsible for the activation of
mTOR signaling in response to resistance exercise, without the need
for IGF-1 activity. A study by O'Neill et al. showed that incubating
isolated muscle cells with PA is sufficient to induce an increase in
mTOR signaling. In the same study, the researchers also showed
that the activation of mTOR by PA occurred even when the isolated
muscle cells had their IGF-1 signaling pathway chemically inhibited-
demonstrating that PA activation of mTOR occurred separately from
IGF-1 signaling. One more interesting finding from this study relates
to the fact that mechanical stress put on the muscle cell from
weightlifting activates the enzyme phospholipase D, which catalyzes
the production of PA.

Since PA increases mTOR-driven muscle protein synthesis, a group
of researchers tested whether PA could also increase muscular size
and strength. In this study by Hoffman et al., they looked at the
influence that PA had on strength in 16 test subjects with adequate weightlifting experience. The subjects were split into two groups, with one group receiving 750 milligrams of PA per day and the other group taking a placebo. During the experiment, each subject lifted weights four days a week at 70 percent of their one-repetition maximum (1RM) for all lifts during the entire eight-week trial period. Each subject was tested for strength and body composition at the end of the experimental period. The results showed that subjects ingesting PA demonstrated a 12.7 percent increase in squat strength and a 2.6 percent increase in muscle mass- while subjects consuming a placebo showed much less improvement in squat strength and virtually no increase in muscle mass.

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